N. Falkenberg

Most-cited papers

1. MiR-221/-222 differentiate prognostic groups in advanced breast cancers and influence cell invasion

   N. Falkenberg, Nataša Anastasov, K. Rappl, H. Braselmann +8 more

   2013British Journal of Cancer61 citations Open access

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2. uPAR enhances malignant potential of triple-negative breast cancer by directly interacting with uPA and IGF1R

   M. Huber, R. Mall, H. Braselmann, A. Feuchtinger +7 more

   2016BMC Cancer38 citations Open access

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3. Three‐dimensional microtissues essentially contribute to preclinical validations of therapeutic targets in breast cancer

   N. Falkenberg, Ines Höfig, M. Rosemann, J. Szumielewski +6 more

   2016Cancer Medicine28 citations Open access

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4. Secreted uPAR isoform 2 (uPAR7b) is a novel direct target of miR-221

   N. Falkenberg, Nataša Anastasov, A. Schaub, Vanja Radulovic +3 more

   2015OncoTarget16 citations Open access

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5. Additive impact of HER2‐/PTK6‐RNAi on interactions with HER3 or IGF‐1R leads to reduced breast cancer progression in vivo

   N. Falkenberg, Nataša Anastasov, Ines Höfig, Ksenia Bashkueva +4 more

   2015Molecular Oncology14 citations Open access

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6. Cyr61 and YB-1 are novel interacting partners of uPAR and elevate the malignancy of triple-negative breast cancer

   M. Huber, N. Falkenberg, S. Hauck, Markus Priller +5 more

   2016OncoTarget13 citations Open access

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7. MEK1 Inhibitor Combined with Irradiation Reduces Migration of Breast Cancer Cells Including miR-221 and ZEB1 EMT Marker Expression

   Nataša Anastasov, E. Hirmer, M. Klenner, J. Ott +7 more

   2020Cancers12 citations Open access

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8. uPA receptor and its interaction partners: Impact as potential therapeutic targets in triple-negative breast cancer.

   M. Aubele, M. Huber, N. Falkenberg, E. Gross +3 more

   2015Journal of Clinical Oncology3 citations

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9. High uPAR and Low miR-221 Expression Predict Poor Disease-Free Survival in Triple-Negative Breast Cancer

   W. Gong, Yueyang Liu, N. Falkenberg, Marion Kiechle +4 more

   2026Pathophysiology0 citations Open access

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10. uPAR enhances malignant potential of triple-negative breast cancer by directly interacting with uPA and IGF1R

    Michaela C. Huber, R. Mall, H. Braselmann, A. Feuchtinger +7 more

    2016BMC Cancer0 citations Open access

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11. 67PThe impact of the uPAR system and its interaction partners as potential therapeutic targets in TNBC

    M. Huber, N. Falkenberg, E. Gross, R. Mall +3 more

    20150 citations Open access

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12. 673: uPAR and its interaction partners: potential new therapy targets in triple negative breast cancer

    M. Huber, N. Falkenberg, M. Schmitt, H. Braselmann +5 more

    20140 citations

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13. 922: Relevance of 3D-microtissues to investigate the therapeutic oncogenes HER2 and PTK6 in breast cancer

    I. Hoefig, N. Falkenberg, M. Rosemann, J. Szumielewski +4 more

    20140 citations

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14. Abstract A046: MiR-221 and -222 expression elevates cell invasion and allows distinction between different prognostic groups in advanced breast cancers

    N. Falkenberg, Nataša Anastasov, H. Braselmann, G. Auer +4 more

    20130 citations

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